Where is Malassezia Furfur found?

Where is Malassezia Furfur found?

Malassezia furfur are opportunistic pathogens that can be found on the skin as normal microbiota. Disease can occur as hypopigmentation or hyperpigmentation on the trunk of humans, in addition to causing dandruff and seborrheic dermatitis.

Is Malassezia Furfur yeast or mold?

Malassezia furfur is an anthropophilic fungus that belongs to the physiological skin flora. The fungus can grow in a yeast phase as well as in a mycelial phase; on nonaffected skin the fungus is mainly prevalent in the yeast phase.

Where does Malassezia grow?

Predisposing factors to Malassezia skin disease include: Humidity. Sweating – hence pityriasis versicolor is common in tropical areas. Oily skin (seborrhoea) – hence it is found mainly on scalp, face and upper trunk.

Where is Malassezia yeast found?

Malassezia yeasts have been found in human dandruff, deep-sea vents, and pretty much everywhere in between. The skin of most if not all warm-blooded animals is covered with these microbes, and while they mostly live in peaceful co-existence with their hosts, they can cause serious diseases in humans and other animals.

What diseases does Malassezia Furfur cause?

Malassezia furfur causes tinea versicolor, catheter-related fungemia, and sometimes pneumonia. Response to either topical or systemic therapy is slow; recovery of granulocyte counts is usually associated with resolution.

Why is Malassezia Furfur important?

Malassezia furfur is a commensal yeast of human skin that has correlates with several common skin conditions. Emerging evidence continually improves the understanding of this microorganism and the diseases it causes.

Is Malassezia Furfur a normal flora?

Malassezia (previously Pityrosporum species) are saprophytic yeasts found on 90% of adults as normal skin flora.

What is the meaning of Malassezia Furfur?

Malassezia furfur (formerly known as Pityrosporum ovale in its hyphal form) is a species of yeast (a type of fungus) that is naturally found on the skin surfaces of humans and some other mammals.

Is Malassezia Furfur dimorphic fungi?

Malassezia furfur is a dimorphic fungus that grows in yeast phase (unaffected skin) and also in mycelial phase (affected skin) [4]. Bacteria and fungi are known to directly or indirectly influence each other’s growth and/or physiology, behavior and survival.

Is Malassezia Furfur normal flora?

Malassezia yeasts are members of normal cutaneous microflora in humans and many domestic and wild animals. As lipophilic fungi, they colonize skin rich in sebaceous glands. They are considered opportunistic pathogens that may cause, under favorable circumstances, various skin diseases.

Is Malassezia Furfur contagious?

furfur is one of a group of common commensal yeasts that normally inhabit human skin but may cause a variety of common dermatologic disorders. These conditions, including SD, PV, and Malassezia folliculitis, are benign and not contagious.

What triggers Malassezia yeast?

The Malassezia (antigen) proteins are found in sweat and the disease is therefore triggered by sweating (sometimes referred to as sweat allergy) (Hiragun et al., 2013; Maarouf et al., 2018).

What is Malassezia furfur?

Malassezia furfur is a member of a monophyletic genus of fungi normally found on human and animal skin. These lipid-dependent, commensal yeasts normally constitute greater than 80% of the total fungal population of human skin and are frequently isolated in both healthy and diseased hosts.[1] 

What causes Malassezia to flare up?

It’s caused when Malassezia furfur population levels grow out of control. First, indoles like pityriarubins impede neutrophils (white-blood cells)that would normally kill the excess Malassezia furfur and start inflammation [3].

What are the treatment options for Malassezia folliculitis?

Systemic antifungal therapy is an option in conjunction with topicals or alone in cases of more extensive involvement or resistant disease. Similar therapy may work for Malassezia folliculitis. [11][15][16] Invasive Malassezia infection warrants prompt removal of central venous catheters due to the yeast’s ability to produce biofilms.